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#36332 In reply to: Pain in the opposite leg after a laminectomy |
Hi,
It’s going on 7 years since my 2 Laminectomy. 4 weeks ago I thought I strain my lower back lifting a 5 pound water bottle. I just got and xray and it shows the following. Can you explain more what’s going on? I had to then get an MRI. waiting on results at the moment.
History: Low back Pain
Technique: Frontal, bilateral oblique and lateral views of the lumbar spine with flexion and extension.
Comparison: 11/-5/2019
Findings:
5 lumbar bodies. New superior endplate concavity of L4 body. Lumbar vertebral bosy hieghts are maintained. Mild multilevel disc height loss. No pathologic motion with flexion or extension.
Impression:
Superior endplate concavity of L4 body, age indeterminate. MRI of the lumbar spine is recommended for futher characterization.
Mild multilevel degeneration changes.Report Ends
#36313Topic: Post cervical surgery pain in forum NECK PAIN |I had a c5-c6 cervical disc replacement. The doctor feels great about the placement etc. it’s 10 months later and I still have pain aggravation in the back of my neck. It’s not intense pain just sore and like a 3 level all day every day, more so at night.
Recently saw a pain doctor and got a CT scan. Curious of your thoughts? Results below.
Results:
EXAM: CT CERVICAL SPINE WITHOUT CONTRAST
History: 42-year-old male with cervical radiculopathy. History of prior disc replacement.
Technique: Helical volume acquisition utilizing 2 mm multiplanar reformats. DLP : 179 mGycm
Comparison: None.
Findings: Spine is imaged from level of the sella through T2. AP alignment shows slight anterolisthesis at C4-5. There is reversal of cervical lordosis. An artificial disc is seen at C5-6 without evidence of periprosthetic bone reabsorption or malpositioning. No fracture, lytic, or blastic lesion is evident. To the extent visible paraspinal soft tissues appear normal.
Craniocervical junction, C1-2: Minimal osteoarthritic changes are seen at the dens C1 articulation of doubtful clinical relevance. These levels are otherwise unremarkable.
C2-3, C3-4: No significant degenerative changes are present.
C4-5: Slight disc space narrowing is present. This level is otherwise unremarkable.
C5-6: Artificial disc has been placed. There is minimal left posterolateral endplate hypertrophic change and left-sided uncovertebral hypertrophy without significant foraminal stenosis. No neural impingement is suspected.
C6-7: No significant degenerative changes are apparent.
C7-T1: Hypertrophic facet changes are noted resulting in mild to moderate bilateral foraminal stenosis, slightly greater on the left than the right (5/32). No definite C8 root impingement is evident although clinical correlation is needed.
T1-2: There is moderate disc space narrowing present. No disc protrusion is visible. The neural foramina are patent.
IMPRESSION: CT Cervical Spine Without Contrast
1. Status post C5-6 artificial disc placement. Position and appearance are normal with no evidence of loosening.2. Potentially significant left C8 foraminal stenosis due to facet arthropathy.
3. No other potential neural compromise is identified.
#36311 In reply to: Advice needed AGAIN!! |Here is my scan from brain:
MRI BRAIN WITHOUT CONTRAST: TECHNIQUE: Multiplanar, multisequence noncontrast brain protocol. Additional series acquired through the brain stem.
INDICATION: Cranial nerve palsy. Dysautonomia. Esophageal motility disorder. Vagus neuropathy.
COMPARISON: None
FINDINGS: Parenchyma: Multiple FLAIR hyperintense foci in the supratentorial white matter, predominantly in a bifrontal distribution. No mass effect or acute infarct. No hemorrhage. Normal appearance of the brainstem.
Parasellar: Normal appearance of the pituitary gland, suprasellar cistern, and cavernous sinuses.
Extra-axial Collection: None
Ventricular System: Normal. No hydrocephalus.
Major Intracranial Flow Voids: Major vascular flow voids appear patent.
Included Orbits: Normal.
Paranasal Sinuses: No significant mucosal thickening. Included nasal cavity and nasopharynx: Unremarkable.Tympanomastoid Cavities: Mastoid and petrous air cells are clear.
Osseous Structures and Soft Tissues: Postsurgical and degenerative changes of the imaged cervical spine. Normal appearance of the bilateral jugular foramina.
#36310Topic: Advice needed AGAIN!! in forum READING X-RAY, MRI & CT SCAN |Hello Dr C. It is your favorite problem child from Kansas!! I hope all is well in Beautoful Hawaii! You are missed here locally for sure! I can’t trust anyone after getting the quality care from you over those rough 3 years!!🥲. But I’m still here and trying to enjoy my grandbabies and life!
My question is that I am having some problems still with my eating. Darn esophagus. But I had a head and neck MRI done last week to see if my vagus nerve was damaged and the MRI of my neck came back as follows:
MRI CERVICAL SPINE WITHOUT CONTRAST: TECHNIQUE: Multiplanar/multisequence cervical spine protocol without contrast. INDICATION: Neck pain. Cervical radiculopathy. Vagus neuropathy. COMPARISON: Cervical spine radiographs from March 11, 2020
FINDINGS: Skull Base: Mild degenerative changes of the atlanto-occipital articulations. Included Intracranial Structures: Normal
Alignment: Straightening of the normal cervical lordosis. No spondylolisthesis.Vertebral Bodies: Interbody fusion and posterior fixation hardware at C3-C4. Disc instrumentation at C5-C6. ACDF at C6-C7. Osseous fusion of the C4-C6 vertebral bodies. Vertebral bodies appear normal in height.
Marrow Signal: Expected background marrow signal. No aggressive osseous lesions.Intervertebral Discs: Marked degenerative endplate changes and disc height loss at C6-C7.
At mild degenerative endplate changes and disc height loss at C7-T1.Spinal Cord: Artifact in the spinal cord at the C3 and C4 levels.
Prominence of the central canal at the C6-T2 levels without an overt syrinx.
Equivocal focal T2 hyperintensities in the bilateral posterior spinal cord at the C1-C2 level (series 100 image 4), although this appearance may be artifactual.
Paraspinal Soft Tissues: Postsurgical changes of the posterior paraspinal soft tissues.
Individual Levels: C2-C3: Facet arthropathy. No spinal canal or neural foraminal stenosis.
C3-C4: Small dorsal endplate osteophytes. Artifact partially obscures the spinal canal. There appears to be mild spinal canal stenosis. No right neural foraminal stenosis. Artifact nearly completely obscures the left neural foramen.
C4-C5: Artifact partially obscures the spinal canal and nearly completely obscures the left neural foramen. No spinal canal or right neural foraminal stenosis.
C5-C6: Facet arthropathy with partial ankylosis of the facet joints. No spinal canal stenosis. Artifact partially obscures the right neural foramen and obscures the left neural foramen. There appears to be mild right neural foraminal stenosis.
C6-C7: Small disc osteophyte complex. Facet arthropathy. No spinal canal stenosis. Artifact partially obscures the neural foramina. There appears to be mild to moderate bilateral neural foraminal stenosis.
C7-T1: Minimal disc osteophyte complex with dorsal annular fissure. Facet arthropathy. No spinal canal or neural foraminal stenosis.Of course no KC Dr wants to see me so they have asked me to contact the amazing surgeon that fused me from c3-T1 for his expertise!!!
#36309Topic: 16kg deadlift 12 weeks after MD op in forum BACK PAIN |Dear Dr Corenman,
I had an L5-S1 microdiscectomy mid-July. My recovery was textbook, and at the 3-month follow up with my surgeon, I told him that the operation had been transformative. (I had experienced periods of nerve firing/twinges, but at around the 10-week mark something happened and I felt great for the first time in 18 months.)
3 days after the surgeon follow-up, I had a physio appointment with my long-time physio during which I was asked to lift a 16kg kettlebell in a ‘deadlift’ position. I intrinsically trust my physio and they have been so helpful prior and post surgery. I am a slight individual (not weak, but not heavily muscled either). Since this lift, I have not been feeling anywhere near as good. I should have refused to do it. My recovery has gone so well, and I would never have wanted to do anything to jeopardise that. So I am angry with myself for following instruction.I now have niggling pains in my gluteus medius and a dull, but not constant, ache in the outer thigh. (Similar symptoms to prior to the surgery, but certainly not as intense – a ‘2 or 3’ on the pain scale rather than an 8 or 9.
This was now 2.5 weeks ago. Should any strain/sprain have gone by now from an over-exerted muscle during that lift? Should I be worried about a reherniation? What would you do in my position?
With many thanks for your time and expertise.#36300Topic: Bertolotti’s Syndrome in Athletes in forum BACK PAIN |My daughter is a collegiate athlete (19) and chasing answers for a very specific back pain caused by a very specific movement. She was diagnosed with Bertolotti’s, but then told it could not be the cause of her pain because a bone scan did not show inflammation at the place where her L5 was hitting. She has had MRI, CT scan and a bone scan.
So my first question would be is it impossible for that area to be causing pain even if no inflammation shows on a bone scan? Is it unreasonable for us to ask to have the area injected to see if it helps? At this point I don’t see what she has to lose since she is facing retirement due to the pain.
She does not have pain in 95% of what she does including daily life and most her skills. She is a gymnast. She has pain related to extreme extension of her lower back and it’s a very specific sharp pain related to only one specific skill. The moment she does this skill she has a sharp, shooting pain that is so excruciating it takes her breath away. The sharp pain is quick because the motion that creates it is quick and then it throbs for a few minutes and then goes away until she is asked to repeat that skill. It feels distinctly to her as if something is being pinched or hitting.
Complicating matters is that she does have a L5 pars fracture. She has had this injury since she was 10 years old, so nearing a decade. She has successfully trained and competed for 10 years with no pain from this injury doing the same skill that is causing the sharp pain. The injury remains unchanged. When she did have pain from the fracture when she was 10 years old it was not anything like the pain she is experiencing now. All training hurt: pounding, landings, all extension, sitting in the car, bending etc. So to her this feels nothing like that. That was an overall dull ache and soreness with all training and this very sharp and specific.
Because she does have a pars fracture the Dr she is seeing did an injection in that area and told her there was no evidence it could be the Bertolotti’s so he would treat the pars fracture. She had 100% zero relief from the injection.
As I said at this point she is facing retirement since she can’t do skills required of her without excruciating pain. I feel we should look further into the Bertolotti’s syndrome even if her current Dr does not believe it can be the cause. Thoughts?
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